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Death From COVID-19 in a Patient Receiving Clozapine: Factors Involved and Prevention Strategies to Consider

Llesuy, Joan Roig; Sidelnik, S Alex
PMID: 32706942
ISSN: 2155-7780
CID: 4535382

Motivational Interviewing on an Addiction Consult Service: Pearls, Perils, and Educational Opportunities [Editorial]

Marcovitz, David E; Sidelnik, S Alex; Smith, Mariah P; Suzuki, Joji
PMID: 32100252
ISSN: 1545-7230
CID: 4444822

[S.l. : Academy of Consultation-Liaison Psychiatry], 2020

Report from NYU Langone Health CL Psychiatry Service

Caravella, Rachel A; Deutch, Allison B; Yee, Michael; Asonye, Uzoamaka; Sidelnik, S Alex; Ying, Patrick; Cruz, Calvin
CID: 4871752

Development of a Virtual Consultation-Liaison Psychiatry Service: A Multifaceted Transformation

Caravella, Rachel A.; Deutch, Allison B.; Noulas, Paraskevi; Ying, Patrick; Liaw, K. Ron-Li; Greenblatt, Jeanne; Collins, Kelsey; Eastburn, H. K.; Fries, Emily; Khan, Shabana; Kozikowski, Adam; Sidelnik, S. Alex; Yee, Michael; Ginsberg, David
ISSN: 0048-5713
CID: 4799202

A Medically Hospitalized Woman with Opioid Use Disorder [Meeting Abstract]

Scimeca, Michael; Suzuki, Joji; Beeder, Ann; Sidelnik, S. Alex; Xiong, Yee; Myles, Angelique
ISSN: 1055-0496
CID: 4444852

Photographing Determinants of Mental Health in Argentina Post-2002 Economic Crisis

Sidelnik, S Alex
PMID: 27259489
ISSN: 1545-7230
CID: 3214632

A Novel Resident-as-Teacher Rotation for Second-Year Psychiatry Residents [Letter]

Vestal, Heather S; Sidelnik, S Alex; Marcovitz, David; Huffman, Jeff
PMID: 26423678
ISSN: 1545-7230
CID: 3214622

Cocaine: A catalyst for human immunodeficiency virus-associated dementia

Dhillon, Navneet K.; Gadgil, Milind; Rahardja, Andy; Callen, Shannon; Sidelnik, Alex; Renfrow, Duncan; Moradi, Amanda; Dhillon, Sukhbir; Kenjale, Himanshu; Kumar, Anil; Buch, Shilpa J.
Injection drug use has been recognized as a major risk factor for AIDS from the outset of the epidemic. Cocaine, one of the most widely abused drugs in the United States can both impair the functions of macrophages & CD4+ lymphocytes and also activate HIV-1 expression in these cells. Cocaine is a multifactorial agent that acts globally to impair the functioning of brain resident cells through multiple pathways. The drug not only promotes virus replication in macrophages, microglia and astrocytes, but can also upregulate CCR5 coreceptor, and reciprocally inhibit its ligands, thereby increasing virus infectivity. Cocaine is known to modulate astroglial function and activation. Cocaine causes a myriad of toxic responses in the neurons: a) it synergizes with viral proteins, Tat and gp120 resulting in exacerbated neuronal apoptosis, b) it causes calcium mobilization and, c) generation of reactive oxygen species. Additionally, cocaine also exerts potent effects on microvascular permeability, thereby impacting the influx of virus-infected inflammatory cells in brain parenchyma. By amplifying the various arms of the toxic responses that characterize HIV-associated dementia (HAD), cocaine skews the balance in favor of the virus leading to accelerated progression and severity of dementia. © 2008 Science Publications.
ISSN: 1553-6203
CID: 4871742

Investigations on four host response factors whose expression is enhanced in X4 SHIV encephalitis

Buch, Shilpa; Sui, Yongjun; Dhillon, Navneet; Potula, Raghava; Zien, Christoher; Pinson, David; Li, Shanping; Dhillon, Sukhbir; Nicolay, Brandon; Sidelnik, Alex; Li, Cicy; Villinger, Tara; Bisarriya, Kunal; Narayan, Opendra
HIV encephalopathy, one of the major complications of HIV infection, involves productive virus replication in macrophages in the brain in association with heightened expression of several host response factors. One or more of these factors are thought to be the cause of the degenerative changes in neurons in the brain. Macaques infected with SIV and SHIV viruses have provided excellent working models for studying mechanisms of the human disease. Although HIV encephalopathy is primarily associated with CCR5-utilizing viruses, our findings have shown that CXCR4-utilizing SHIVs were also capable of causing the syndrome in rhesus macaques. In SHIV-infected macaques, approximately 30% of the animals developed encephalitis. In order to understand the factors leading to end-stage encephalitis, we performed microarray analyses on brains of encephalitic and non-encephalitic-infected macaques, and found pronounced enhancement of expression of interleukin-4, platelet-derived growth factor-B chain, monocyte chemoattractant protein-1 and CXCL10 in the brains of the encephalitic animals. This review discusses the role of each of these factors in mediating SHIV encephalitis.
PMID: 15579283
ISSN: 0165-5728
CID: 4444842