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Highlights of January '97 - March '97 television placements

Chu, Benjamin; Colvin, Stephen B.; Llinas, Rudolfo R.; Menche, David; Weiss, Edwin
Extent: 1 videocassette (6 min.) : sd., col. ; 1/2 in
ISBN: n/a
CID: 726

False positive signal-averaged electrocardiogram produced by atrial flutter [Case Report]

Schrem SS; Nachamie M; Weiss E
PMID: 2389709
ISSN: 0002-8703
CID: 64555

Right atrial papillary fibroelastoma: diagnosis by transthoracic and transesophageal echocardiography and percutaneous transvenous biopsy [Case Report]

Schwinger ME; Katz E; Rotterdam H; Slater J; Weiss EC; Kronzon I
PMID: 2816690
ISSN: 0002-8703
CID: 10431

The usefulness of echocardiography in a long term health care facility

Kronzon, I; Freedberg, R S; Khan, B; Cohen, M L; Berger, A; Weiss, E C
PMID: 3136411
ISSN: 0028-7628
CID: 100110

Atrial natriuretic peptide administration to normal and salt depleted rats--effects on digoxin-like immunoreactive factor, aldosterone, ACTH, and renal function

Shilo, L; Pomeranz, A; Rathaus, M; Weiss, E; Bernheim, J; Shenkman, L
In view of the known interrelationships between renin, aldosterone, and atrial natriuretic peptide (ANP), we sought to examine whether there also exists an interaction between ANP and digoxin-like immunoreactive factor (DLIF). We therefore studied the effects of ANP administration on normal and salt-depleted rats, and measured the effects on blood pressure, urine output, glomerular filtration rate, sodium excretion, aldosterone, ACTH, and DLIF levels. ANP administration resulted in a significant elevation of sodium excretion and glomerular filtration rate and a fall in blood pressure. DLIF concentrations in plasma rose significantly, as did urinary DLIF excretion. ANP administration resulted in a fall in aldosterone as well as ACTH. These observations suggest that ANP has a direct inhibitory effect on ACTH secretion. Our findings support the concept of an interrelationship between ANP and DLIF.
PMID: 2835565
ISSN: 0024-3205
CID: 545462

Anesthetic and supportive management during experimental pulsatile flow perfusion studies in calves

Short, C E; Harvey, R C; Fisher, F E; Cunningham, J N Jr; Rose, D M; Gelbfish, J; Weiss, E; Grossi, E
The purpose of this study was to determine the factors influencing successful experimental cardiopulmonary bypass studies using pulsatile flow perfusion and the medications and methodology necessary to produce successful bypass in calves. In six calves showing no cardiopulmonary pathology prior to bypass procedures, successful anesthesia and surgical intervention was accomplished. Animals were maintained on 5 hours of pulsatile flow bypass perfusion. Successful recovery from the procedures was accomplished. In two calves with pre-existing pulmonary pathology, anesthetic and surgical intervention was accomplished with the utilization of extensive anesthetic management and cardiac supportive medications until the animals could be initiated into 5 hours of pulsatile flow bypass perfusion, in spite of major pulmonary dysfunction. In these two animals, attempts to resuscitate upon termination of pulsatile flow perfusion were unsuccessful due to pre-existing excessive lesions in the lungs. This study shows a contrast between complete success of a pulsatile flow system in normal subjects versus the ultimate failure in experimental animals with pre-existing pulmonary pathology. The inability of experimental calves with a diseased lung to resume spontaneous cardiopulmonary function after the challenges of thoracic intervention indicates the unsuitability of animals with marked pre-existing pulmonary disease status for use in cardiopulmonary bypass studies
PMID: 3586615
ISSN: 0023-6764
CID: 126724

The polyglandular deficiency syndrome: a new variant in Persian Jews [Case Report]

Shapiro, M S; Zamir, R; Weiss, E; Radnay, J; Shenkman, L
Five Persian Jews were detected with the polyglandular deficiency syndrome (PDS). Primary hypoparathyroidism and hypogonadism were present in each, adrenal insufficiency in two, and insulin-dependent diabetes mellitus and latent hypothyroidism in single subjects. The percentage of T and B cells, and the mononuclear cell response to phytohemagglutinin and Concanavalin A were normal in all five. IgG and IgA levels and the OKT4+/OKT8+ cell ratio were low in one subject. Antinuclear and antithyroid antibodies were present in one subject. HLA-DR5 was present in 4/4, HLA-24 and B5 (B51) in 3/4 subjects. A single case of isolated hypoparathyroidism (IHP) was detected among 12 first degree relatives. HLA antigens B8, DR3, were absent in all of these subjects. Seven non-Iranian Jews with IHP were also examined. HLA A26 or A25 were present in all seven. Persian Jews appear to have a unique variant of PDS.
PMID: 3496374
ISSN: 0391-4097
CID: 545542

Gonadotropin, prolactin and TSH secretion in patients with myasthenia gravis

Shapiro, M S; Weiss, E; Kott, E; Taragan, R; Shenkman, L
Hypothalamic pituitary function was studied in 13 patients with myasthenia gravis. Gonadotropin, TSH, and prolactin dynamics were investigated using conventional provocative stimuli. No consistent abnormality was found in either gonadotropin or prolactin release. Abnormal TSH responses to TRH administration was present in six of the 13 patients in association with normal free thyroxine indices and the absence of antithyroid antibodies. This latter observation is relevant when the association of myasthenia gravis with hyperthyroidism, thyroiditis and hypothyroidism is considered.
PMID: 6442309
ISSN: 0391-4097
CID: 545622

Case report: echocardiographic observations in patients with Friedreich's ataxia

Weiss, E; Kronzon, I; Winer, H E; Berger, A R
Echocardiography was performed on 11 patients with Friedreich's Ataxia. Eight of 11 had asymmetric septal hypertrophy and systolic anterior motion of the anterior leaflet of the mitral valve at rest or after inhalation of amyl nitrite. Two patients had concentric left ventricular hypertrophy. In view of this high incidence of hypertrophic cardiomyopathy, echocardiography is suggested as part of the routine evaluation of the patient with Friedreich's Ataxia
PMID: 7198380
ISSN: 0002-9629
CID: 100144


ISSN: 0012-3692
CID: 40194