Faculty Bibliography

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school:SOM

Department/Unit:Cell Biology

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14178

Proceedings (American Association for Cancer Research). 2003:44(6):247-247.DOI:

Coating of culture plates with collagen increases the in vitro effects of both PS-341 and ionizing radiation (RT) [Meeting Abstract]

Ng, Bruce; Snell, Jamaal; Brooks, Peter; Liebes, Leonard; Formenti, Silvia
BIOSIS:PREV200300474897
ISSN: 0197-016x
CID: 109234

The burden of musculoskeletal conditions at the start of the new millennium

Agel, J; Akesson, K; Amadio, PC; Anderson, M; Badley, E; Balint, G; Bellamy, N; Bigos, S; Bishop, N; Bivans, B; Bjorke, PA; Brooks, P; Browner, BD; Buckwalter, J; Callahan, L; Chahade, WH; Chopra, A; Cimmino, M; Cooper, C; Darmawan, J; De, Mesquita, KC; De, Smedt, M; Delmas, P; Dequeker, J; Dieppe, P; Dougados, M; Dreinhofer, KE; Ehrlich, GE; Gans, BM; Genant, HK; Grob, D; Guillemin, F; Hamelynk, K; Hazan, E; Hazes, JM; Hochberg, MC; Johnell, O; Kanis, J; Kinasha, AD; Kuder, AU; Lau, EMC; Lawrence, RC; Lidgren, L; Lips, P; Lohmander, S; Luchter, S; Mackenzie, EJ; Marini, JC; Melton, J; Mithal, A; Mock, C; Mohan, D; Moser, R; Nassonova, VA; Nordin, M; Oestern, H-J; Pattison, D; Petty, RE; Poor, G; Rasker, JJH; Raspe, H; Reginster, JY; Rizzoli, R; Sethi, D; Shanmugasundaram, TK; Shewan, CM; Shichikawa, K; Stucki, G; Symmons, D; Van, Der, Linden, S; Vischer, TL; Walsh, NE; Weinstein, SL; Woolf, AD; Yelin, E; Yoshizawa, H
Geneva : World Health Organization, 2003
Extent: x, 218 p.
ISBN:
CID: 1778492

Biocomputing : computer tools for biologists

Brown, Stuart M
Westborough, MA US : Eaton Pub., 2003
Extent: ix, 539 p. ; 26 cm.
ISBN: 9781881299172
CID: 1671542
FASEB journal. 2003:17(4):A437-A437.DOI:

Influence of altered metabolic pathways on fiber-type specific gene expression in C2C12 cells. [Meeting Abstract]

Teissedre, BDC; Au, N; Swoap, SJ
ISI:000181733102070
ISSN: 0892-6638
CID: 2159352

Endoplasmic Reticulum Stress Responses

Chapter by: Ron, D
in: Handbook of Cell Signaling by Bradshaw RA; Dennis EA [Eds]
San Diego, Calif. : Academic Press, c2003
pp. 359-363
ISBN: 9780080533575
CID: 1843352

Endoplasmic Reticulum Stress Responses

Chapter by: Ron, D
in: Handbook of Cell Signaling by Bradshaw RA; Dennis EA [Eds]
San Diego, Calif. : Academic Press, c2003
pp. 263-267
ISBN: 9780080533575
CID: 1843342

Neurotrophin Signaling in Development

Chapter by: Kim, AH; Chao, MV
in: Handbook of Cell Signaling by
pp. 839-843
ISBN: 9780080533575
CID: 1843362
Developmental biology (Orlando). 2003:259(2):585-585.DOI:

Analysis of fear of intimacy: a novel gene involved in gonad formation. [Meeting Abstract]

Mathews, W; Van Doren, M
ISI:000184373300574
ISSN: 0012-1606
CID: 2206522

Annexins and tissue mineralization -- matrix vesicles, ion channel activity of annexins and anexin V-collagen interactions

Chapter by: Kirsch, T
in: Annexins : biological importance and annexin-related pathologies by Bandorowicz-Pikula, Joanna [Eds]
Georgetown TX : Landes Bioscience/Eurekah.com, 2003
pp. 172-181
ISBN: 030647834x
CID: 4803
Current opinion in orthopaedics. 2003:14(5):356-361.DOI:

Osteoarthritis: A cellular differentiation defect?

Kirsch T.
Purpose of review: Osteoarthritis is the most common form of arthritis, affecting a large population of mostly elderly people. No cure for osteoarthritis currently exists. Ultimate treatment is joint replacement. Understanding the mechanisms causing onset and progression is critical. This review describes recent findings that provide new insights into changes of cellular phenotype in osteoarthritis as a possible reason for tissue failure. Recent findings: Recent findings suggest that articular chondrocytes, when stimulated, can undergo hypertrophic and terminal differentiation events similar to those occurring during endochondral bone formation. Interestingly, collagenase-3 (matrix metalloproteinase-13), a main matrix-degrading enzyme in osteoarthritis, is expressed only in terminally differentiated chondrocytes during normal development. Summary: Although terminal differentiation events are required for endochondral bone formation, they lead to cartilage destruction when occurring in articular chondrocytes. Maintaining the articular chondrocyte phenotype and preventing these cells from undergoing hypertrophic and terminal differentiation might provide novel therapeutic targets to prevent onset or progression of osteoarthritis
EMBASE:2003371101
ISSN: 1041-9918
CID: 83072